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it seem to say that the study says that the condition is linked to a defect in the genome which prevents testosterone to dock to the recipients and therefore not being effective
That sounds like research on the androgen receptor—the protein through which testosterone acts—but “a genetic defect prevents testosterone from docking” is stronger than what the transgender study established.[1] I don’t know whether this i A likely match is Hare and colleagues’ 2009 paper, Androgen Receptor Repeat...
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- That sounds like research on the androgen receptor—the protein through which testosterone acts—but “a genetic defect prevents testosterone from docking” is stronger than what the transgender study established.[1] I don’t know whether this i
- A likely match is Hare and colleagues’ 2009 paper, Androgen Receptor Repeat Length Polymorphism Associated with Male to Female Transsexualism (using the terminology of its time).
- It investigated variation in the androgen receptor gene in transgender women.[1] Its proposed explanation was reduced androgen signaling as a possible contributor to gender identity—not proof that testosterone cannot bind, becomes completel
- That should not be confused with a general explanation for transgender identity.[13] Can you share the link, screenshot, or exact wording?
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openai.comCreate a landscape editorial hero image for this Studio Global article: it seem to say that the study says that the condition is linked to a defect in the genome which prevents testosterone to dock to the recipie. Article summary: That sounds like research on the androgen receptor—the protein through which testosterone acts—but “a genetic defect prevents testosterone from docking” is stronger than what the transgender study established.[1] I don’t. Topic tags: general web, javascript, health, education. Style: premium digital editorial illustration, source-backed research mood, clean composition, high detail, modern web publication hero. Use reference image context only for broad subject, composition, and topical grounding; do not copy the exact image. Avoid: logos, brand marks, copyrighted characters, real person likenesses, fake screenshots, UI text, readable text, watermarks, charts w
That sounds like research on the androgen receptor—the protein through which testosterone acts—but “a genetic defect prevents testosterone from docking” is stronger than what the transgender study established.1 I don’t know whether this is the exact paper you heard about, or whether the Johns Hopkins attribution is correct.
- A likely match is Hare and colleagues’ 2009 paper, Androgen Receptor Repeat Length Polymorphism Associated with Male-to-Female Transsexualism (using the terminology of its time). It investigated variation in the androgen-receptor gene in transgender women.
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- Its proposed explanation was reduced androgen signaling as a possible contributor to gender identity—not proof that testosterone cannot bind, becomes completely ineffective, or that one genetic defect explains being transgender.
- A different condition, androgen insensitivity syndrome, does involve androgen-receptor mutations that impair the body’s response to testosterone. That should not be confused with a general explanation for transgender identity.
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Can you share the link, screenshot, or exact wording? That would let us distinguish the receptor-gene hypothesis from androgen insensitivity syndrome.
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What is the short answer to "it seem to say that the study says that the condition is linked to a defect in the genome which prevents testosterone to dock to the recipients and therefore not being effective"?
That sounds like research on the androgen receptor—the protein through which testosterone acts—but “a genetic defect prevents testosterone from docking” is stronger than what the transgender study established.[1] I don’t know whether this i
What are the key points to validate first?
That sounds like research on the androgen receptor—the protein through which testosterone acts—but “a genetic defect prevents testosterone from docking” is stronger than what the transgender study established.[1] I don’t know whether this i A likely match is Hare and colleagues’ 2009 paper, Androgen Receptor Repeat Length Polymorphism Associated with Male to Female Transsexualism (using the terminology of its time).
What should I do next in practice?
It investigated variation in the androgen receptor gene in transgender women.[1] Its proposed explanation was reduced androgen signaling as a possible contributor to gender identity—not proof that testosterone cannot bind, becomes completel
Sources
pubmed.ncbi.nlm.nih.govSimilar articles for PMID: 18962445 - Search Results - PubMed
link.springer.comHigh-resolution functional mapping of androgen receptor variants - Nature Biomedical Engineering